קטגוריה: המלצות תזונה ומתכונים על פי תזונת אומגה
רצועות סינטה וירקות
עור יבש- הפתרונות הטבעיים
במסווה של גזר גמדי
חשמל זורם בכפות רגליך
גזור ושמור- רשימה תליה על המקרר
תזונת פליאו ותזונת אומגה
לחיות ללא תרופות
- המחסור באומגה 3
- עודף באומגה 6
- עודף סוכר ועמילן
- כיצד ניתן לחיות ללא תרופות על ידי תזונה נכונה
פרוקטוז סוכר פירות גורם מחלות
Potential role of sugar (fructose) in the epidemic of hypertension, obesity and the metabolic syndrome, diabetes, kidney disease, and cardiovascular disease Richard J Johnson, Mark S Segal, Yuri Sautin, Takahiko Nakagawa, Daniel I Feig, Duk-Hee Kang, Michael S Gersch, Steven Benner, and Laura G Sa´nchez-Lozada
ABSTRACT Currently, we are experiencing an epidemic of cardiorenal disease characterized by increasing rates of obesity, hypertension, the metabolic syndrome, type 2 diabetes, and kidney disease. Whereas excessive caloric intake and physical inactivity are likely important factors driving the obesity epidemic, it is important to consider additional mechanisms.
We revisit an old hypothesis that sugar, particularly excessive fructose intake, has a critical role in the epidemic of cardiorenal disease. We also present evidence that the unique ability of fructose to induce an increase in uric acid may be a major mechanism by which fructose can cause cardiorenal disease.
Finally, we suggest that high intakes of fructose in African Americans may explain their greater predisposition to develop cardiorenal disease, and we provide a list of testable predictions to evaluate this hypothesis.
Am J Clin Nutr 2007;86:899 –906.
סוכר הורג
Hyperinsulinemic diseases of civilization: more than just Syndrome X Loren Cordain*, Michael R. Eades, Mary D. Eades Department of Health and Exercise Science, Colorado State University, Fort Collins, CO 80523, USA Received 27 June 2002; received in revised form 23 December 2002; accepted 3 January 2003
Abstract Compensatory hyperinsulinemia stemming from peripheral insulin resistance is a well-recognized metabolic disturbancethat is at the root cause of diseases and maladies of Syndrome X (hypertension, type 2 diabetes, dyslipidemia, coronary artery disease, obesity, abnormal glucose tolerance).
Abnormalities of fibrinolysis and hyperuricemia also appear to be members of the cluster of illnesses comprising Syndrome X. Insulin is a well-established growth-promoting hormone, and recent evidence indicates that hyperinsulinemia causes a shift in a number of endocrine pathways that may favor unregulated tissue growth leading to additional illnesses.
Specifically, hyperinsulinemia elevates serum concentrations of free insulin-like growth factor-1 (IGF-1) and androgens, while simultaneously reducing insulin-like growth factor-binding protein 3 (IGFBP-3) and sex hormone-binding globulin (SHBG).
Since IGFBP-3 is a ligand for the nuclear retinoid X receptor a, insulin-mediated reductions in IGFBP-3 may also influence transcription of anti-proliferative genes normally activated by the body’s endogenous retinoids.
These endocrine shifts alter cellular proliferation and growth in a variety of tissues, the clinical course of which may promote acne, early menarche, certain epithelial cell carcinomas, increased stature, myopia, cutaneous papillomas (skin tags), acanthosis nigricans, polycystic ovary syndrome (PCOS) and male vertex balding.
Consequently, these illnesses and conditions may, in part, have hyperinsulinemia at their root cause and therefore should be classified among the diseases of Syndrome X. 2003 Elsevier Science Inc. All rights reserved.
Keywords: Acne; Early menarche; Epithelial cell carcinomas; Hyperinsulinemia; Increased stature; Myopia; Cutaneous papillomas (skin tags); Acanthosis nigricans; Polycystic ovary syndrome; Male vertex balding.
גם סוכר פירות הורג
Grams of Fructose |
Serving Size |
Fruit |
4.6 |
1 cup |
Boysenberries |
4.8 |
orange1 |
Tangerine/mandarin medium |
5.4 |
1 medium |
Nectarine |
5.9 |
1 medium |
Peach |
6.1 |
1 medium |
Orange (navel) |
6.3 |
1/2 medium |
Papaya |
6.7 |
1/8 of med. melon |
Honeydew |
7.1 |
1 medium |
Banana |
7.4 |
1 cup |
Blueberries |
7.7 |
1 medium |
Date (Medjool) |
9.5 |
1 medium |
Apple (composite) |
10.6 |
1 medium |
Persimmon |
11.3 |
1/16 med melon |
Watermelon |
11.8 |
1 medium |
Pear |
12.3 |
1/4 cup |
Raisins |
12.4 |
1 cup |
Grapes, seedless (green or red) |
16.2 |
1/2 medium |
Mango |
16.4 |
1 cup |
Apricots, dried |
23.0 |
1 cup |
Figs, dried |
0 |
1 medium |
Limes |
0.6 |
1 medium |
Lemons |
0.7 |
1 cup |
Cranberries |
0.9 |
1 medium |
Passion fruit |
1.2 |
1 medium |
Prune |
1.3 |
1 medium |
Apricot |
2.2 |
2 medium |
Guava |
2.6 |
1 medium |
Date (Deglet Noor style) |
2.8 |
1/8 of med. melon |
Cantaloupe |
3.0 |
1 cup |
Raspberries |
3.4 |
1 medium |
Clementine |
3.4 |
1 medium |
Kiwifruit |
3.5 |
1 cup |
Blackberries |
3.6 |
1 medium |
Star fruit |
3.8 |
10 |
Cherries, sweet |
3.8 |
1 cup |
Strawberries |
4.0 |
1 cup |
Cherries, sour |
4.3 |
1/2 medium |
Grapefruit, pink or red |
כיצד דגנים גורמים למחלות אוטואימוניות
Surprises from Celiac Disease
Study of a potentially fatal food-triggered disease has uncovered a process that may contribute to many autoimmune disorders
By Alessio Fasano
שוקולד וקקאו
Review Article
Cocoa and health: a decade of research
Karen A. Cooper1, Jennifer L. Donovan2, Andrew L. Waterhouse3 and Gary Williamson1* 1Nestle´ Research Center, Vers-Chez-les-Blanc, PO Box 44, CH-1000 Lausanne 26, Switzerland 2Department of Psychiatry and Behavioural Sciences, Medical University of South Carolina, Charleston, SC 29425, USA 3Department of Viticulture & Enology, University of California, Davis, CA 95616, USA (Received 5 December 2006 – Revised 29 May 2007 – Accepted 31 May 2007)
It has been over 10 years since the first mention in a medical journal about cocoa and chocolate as potential sources of antioxidants for health.
During this time, cocoa has been found to improve antioxidant status, reduce inflammation and correlate with reduced heart disease risk; with these results, and its popularity, it has received wide coverage in the press
However, after 10 years of research, what is known about the potential health benefits of cocoa and what are the important next steps in understanding this decadent source of antioxidants?
Cocoa: Chocolate: Health: Polyphenols: Antioxident
תזונה אומגה 3 ואוטיזם
ריזוטו ירקות שורש- ללא אורז
מקארל ברוטב
לברק אפוי בתנור- מאת רעי בן-צבי
אומגה 3 משפרת ביצועי ספורטאים ומונעת מחלות ספורט
Omega-3 Fatty Acids and Athletics Artemis P. Simopoulos, MD Current Sports Medicine Reports 2007, 6:230–236 Current Medicine Group LLC ISSN 1537-890x
Human beings evolved consuming a diet that contained about equal amounts of n-6 and n-3 essential fatty acids. Today, in Western diets, the ratio of n-6 to n-3 fatty acids ranges from approximately 10:1 to 20:1 instead of the traditional range of 1:1 to 2:1.
Studies indicate that a high intake of n-6 fatty acids shifts the physiologic state to one that is prothrombotic and proaggregatory, characterized by increases in blood viscosity, vasospasm, and vasoconstriction, and decreases in bleeding time. ?-3 fatty acids, however, have anti-inflammatory, antithrombotic, antiarrhythmic, hypolipidemic, and vasodilatory properties.
Excessive radical formation and trauma during high-intensity exercise leads to an inflammatory state that is made worse by the increased amount of n-6 fatty acids in Western diets, although this can be counteracted by eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA).
For the majority of athletes, especially those at the leisure level, general guidelines should include EPA and DHA of about 1 to 2 g/d at a ratio of EPA:DHA of 2:1.
חיסונים ותמותת תינוקות
ביצועים ספורטיבים וויטמין D
Athletic Performance and Vitamin D JOHN J. CANNELL1, BRUCE W. HOLLIS2, MARC B. SORENSON3, TIMOTHY N. TAFT4, and JOHN J. B. ANDERSON5
1Atascadero State Hospital, Atascadero, CA; 2Departments of Biochemistry and Molecular Biology, Medical University of South Carolina, Charleston, SC; 3sunlightandhealth.org, Saint George, UT; 4Departments of Orthopedics and Sports Medicine, University of North Carolina, Chapel Hill, NC; and 5Departments of Public Health and Nutrition, University of North Carolina, Chapel Hill, NC
ABSTRACT
CANNELL, J. J., B. W. HOLLIS, M. B. SORENSON, T. N. TAFT, and J. J. ANDERSON.
Athletic Performance and Vitamin D. Med.Sci. Sports Exerc., Vol. 41, No. 5, pp. 1102–1110, 2009.
Purpose: Activated vitamin D calcitriol) is a pluripotent pleiotropic secosteroid hormone. As a steroid hormone, which regulates more than 1000 vitamin D–responsive human genes, calcitriol may influence athletic performance. Recent research indicates that intracellular calcitriol levels in numerous human tissues, including nerve and muscle tissue, are increased when inputs of its substrate, the prehormone vitamin D, are increased.
Methods: We reviewed the world’s literature for evidence that vitamin D affects physical and athletic performance.
Results: Numerous studies, particularly in the German literature in the 1950s, show vitamin D–producing ultraviolet light improves athletic performance. Furthermore, a consistent literature indicates physical and athletic performance is seasonal; it peaks when 25-hydroxy-vitamin D [25(OH)D] levels peak, declines as they decline, and reaches its nadir when 25(OH)D levels are at their lowest. Vitamin D also increases the size and number of Type II (fast twitch) muscle fibers. Most cross-sectional studies show that 25(OH)D levels are directly associated with musculoskeletal performance in older individuals. Most randomized controlled trials, again mostly in older individuals, show that vitamin D improves physical performance.
Conclusions: Vitamin D may improve athletic performance in vitamin D–deficient athletes. Peak athletic performance may occur when 25(OH)D levels approach those obtained by natural, full-body, summer sun exposure, which is at least 50 ngImLj1. Such 25(OH)D levels may also protect the athlete from several acute and chronic medical conditions.
Key Words: PHYSICAL PERFORMANCE, PEAK ATHLETIC PERFORMANCE, ACTIVATED VITAMIN D, CALCITRIOL, 1, 25-DIHYDROXY-VITAMIN D, 25(OH)D
שומן רווי נגד סרטן- תוצאות מחקר
a b s t r a c t
Purpose: Epidemiologic and experimental studies suggest that dietary fat intake may affect risk of pancreatic cancer, but published results are inconsistent.
Methods: We examined risk associations for specific types of dietary fat intakes and related food sources among 111,416 participants in the Prostate, Lung, Colorectal and Ovarian Cancer Screening Trial. We used Cox proportional hazards regression to examine associations between fat intake and pancreatic cancer risk.
Results: Over a mean 8.4 years of follow-up, 411 pancreatic cancer cases were identified. We observed an inverse association between saturated fat intake and pancreatic cancer risk (hazard ratio [HR], 0.64 comparing extreme quintiles; 95% confidence interval [CI], 0.46e0.88), but the association became weaker and nonsignificant when individuals with fewer than 4 years of follow-up were excluded to avoid possible reverse causation (HR, 0.88; 95% CI, 0.58e1.33). Total fat intake showed a similar pattern of association, whereas intakes of monounsaturated and polyunsaturated fats and fats from animal or plant sources showed no associations with risk.
Conclusions: These results do not support the hypothesis of increased pancreatic cancer risk with higher fat consumption overall or by specific fat type or source. Dietary changes owing to undetected disease may explain the observed inverse association with saturated fat.
In conclusion, our findings do not support a positive association between dietary fat intake and risk of pancreatic cancer. Instead we observed an inverse association that was attenuated with exclusion of cases with shorter term follow-up. Our results highlight the need to carefully examine possible reverse causation in studies of diet and pancreatic malignancy and the importance of basing conclusions on a body of evidence from studies with longer term follow-up.
פרוקטוז- טבלאות חישוב והסברים
השפעה של אורז מלא על סכרתיים
Effects of the brown rice diet on visceral obesity and endothelial function: the BRAVO study. Shimabukuro M, Higa M, Kinjo R, Yamakawa K, Tanaka H, Kozuka C, Yabiku K, Taira SI, Sata M, Masuzaki H.
Source
Department of Cardio-Diabetes Medicine, The University of Tokushima Graduate School of Health Biosciences, 3-18-15 Kuramoto, Tokushima 770-8503, Japan.
Abstract
Brown rice (BR) and white rice (WR) produce different glycaemic responses and their consumption may affect the dietary management of obesity. In the present study, the effects of BR and WR on abdominal fat distribution, metabolic parameters and endothelial function were evaluated in subjects with the metabolic syndrome in a randomised cross-over fashion. In study 1, acute postprandial metabolic parameters and flow- and nitroglycerine-mediated dilation (FMD and NMD) of the brachial artery were determined in male volunteers with or without the metabolic syndrome after ingestion of either BR or WR. The increases in glucose and insulin AUC were lower after ingestion of BR than after ingestion of WR (P= 0·041 and P= 0·045, respectively). FMD values were decreased 60 min after ingestion of WR (P= 0·037 v. baseline), but the decrease was protected after ingestion of BR. In study 2, a separate cohort of male volunteers (n 27) with the metabolic syndrome was randomised into two groups with different BR and WR consumption patterns. The values of weight-based parameters were decreased after consumption of BR for 8 weeks, but returned to baseline values after a WR consumption period. Insulin resistance and total cholesterol and LDL-cholesterol levels were reduced after consumption of BR. In conclusion, consumption of BR may be beneficial, partly owing to the lowering of glycaemic response, and may protect postprandial endothelial function in subjects with the metabolic syndrome. Long-term beneficial effects of BR on metabolic parameters and endothelial function were also observed.
כללי החיים ללא תרופות
ארוחה לפי תזונת אומגה
ארוחת פשטידות
שיפודי בשר לא חרוך
ארוחת שבת
פשטידת ברוקולי וגבינה
פשטידת תרד בלי קמח בכלל
קינוח
ארוחה אותנטית בגליל
סלט עדשים שחורים
מתאבנים
פשטידת ביצה
ארוחת יום שישי
האמת על קרם הגנה
תזונה קטוגנית אינה גורמת לנזק בכליות באנשים שמנים בריאים
תזונה קטוגנית
A Review of Low-carbohydrate Ketogenic Diets Eric C.Westman,MD,MHS,John Mavropoulos,MPH, William S.Yancy,Jr.,MD,MHS,and Jeff S.Volek,PhD,RD Current Atherosclerosis Reports 2003
Introduction Obesity has been implicated as the second leading preventable cause of death in the United States, and studies support that intentional weight loss leads to a reduction in overall mortality [1,2]. In response to the emerging epidemic of obesity, there has been a renewal of interest in alternative diets. Given the current unfavorable trends with conventional approaches, a reconsideration of previously unevaluated alternative diet therapies is not unreasonable. Based on lay-press book sales, the most popular alternative weight-loss diet is the very low-carbohydrate diet. Diets that limit carbohydrate intake have been called “low-carbohydrate,” “very-low-carbohydrate,” “high-protein,” “high-fat,” and “ketogenic.” Presently, there is no consensus on a precise quantitative definition for a low-carbohydrate diet. A lowcarbohydrate diet may or may not be a “high-protein diet” depending upon the food choice and caloric intake. For the purpose of this review, we define a “low-carbohydrate ketogenic diet” (LCKD) as daily consumption of fewer than 50 g of carbohydrate, regardless of fat, protein, or caloric intake.
Conclusions In controlled trials, the LCKD has been demonstrated to lead to weight loss and improvements in fasting triglycerides, HDL cholesterol, and cholesterol/HDL ratio over a 6-month period. Clinical trials assessing the long-term safety and effectiveness of the LCKD are needed. The LCKD needs to be evaluated not only for obesity, but also for conditions that have a theoretical basis for improvement by a reduction in dietary carbohydrate and a shift from a glucocentric to adipocentric metabolism. Although the basic physiology of an LCKD resembles the state of prolonged fasting, there are key differences such that basic studies regarding LCKD physiology are urgently needed. Fundamental questions regarding fuel utilization and the regulation of gluconeogenesis and ketogenesis in the presence of protein and fat intake need to be addressed. The cultural example of the Inuit demonstrates the remarkable adaptability of the human organism to withstand extremes of macronutrient intake, necessitating the questioning of whether dietary carbohydrate is required for human function [74]. Because these findings from clinical trials have been counterintuitive, clinical research strongly suggests that studying the LCKD may lead to unexpected advances in molecular cell biology and clinical therapeutics.
תזונה קטוגנית מונעת אפילפסיה
Do Patients With Absence Epilepsy Respond to Ketogenic Diets? Laura B. Groomes1, Paula L. Pyzik, BA1, Zahava Turner, RD1, Jennifer L. Dorward, RD1, Victoria H. Goode, MLIS1, and Eric H. Kossoff, MD
Abstract Dietary therapies are established as beneficial for symptomatic generalized epilepsies such as Lennox-Gastaut syndrome; however, the outcome for idiopathic generalized epilepsy has never been specifically reported. The efficacy of the ketogenic and modified Atkins diet for childhood and juvenile absence epilepsy was evaluated from both historical literature review and patients treated at Johns Hopkins Hospital. Upon review of 17 published studies in which absence epilepsy was included as a patient subpopulation, approximately 69% of 133 with clear outcomes patients who received the ketogenic diet had a >50% seizure reduction, and 34% of these patients became seizure free. At Johns Hopkins Hospital, the ketogenic diet (n =8) and modified Atkins diet (n =13) led to similar outcomes, with 18 (82%) having a >50% seizure reduction, of which 10 (48%) had a >90% seizure reduction and 4 (19%) were seizure free. Neither age at diet onset, number of anticonvulsants used previously, particular diet used, nor gender correlated with success. In summary, both the ketogenic and modified Atkins diets appear to be effective treatments for intractable absence epilepsy. Not only were a significant majority of patientsimproved, but many had periods of seizure freedom. Further prospective studies of diets for absence epilepsy are warranted.